Apoptosis in acute renal failure
نویسندگان
چکیده
منابع مشابه
Acute Renal Failure Following Brake Fluid Poisoning
Brake fluid is a mixture of different Glycol derivatives which are poisoning and their ingestion triggers significant CNS symptoms, severe metabolic acidosis, cardiac, pulmonary, and renal failure. Here in this study, a case is reported who had been ingested 150mL of brake fluid for suicidal attempt.
متن کاملAcute renal failure following brake fluid poisoning
Brake fluid is a mixture of different Glycol derivatives which are poisoning and their ingestion triggers significant CNS symptoms, severe metabolic acidosis, cardiac, pulmonary, and renal failure. Here in this study, a case is reported who had been ingested 150mL of brake fluid for suicidal attempt.
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BACKGROUND Klotho is associated with the suppression of several ageing phenotypes. Because high klotho gene expression was detected in the kidney and several studies have found altered expression in animal models, we explored the physiological relevance of klotho expression in the kidney under renal ischemia reperfusion injury (IRI). METHODS Male Wistar rats were subjected to bilateral renal ...
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Acute renal failure (ARF) is a common condition in hospitalized patients. The aetiology and physiological characteristics differ from those of chronic renal failure (CRF) and both conditions should be approached differently. At present, the approach to the management of ARF is very heterogeneous. Attempts have therefore been made to improve consensus and to standardize treatment in the Acute Di...
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Adequate nutritional support is necessary to maintain protein stores and to correct pre-existing or disease-related deficits in lean body mass. The objectives for nutritional support for patients with acute renal failure (ARF) are not much different from those with other catabolic conditions. The principles of nutritional support for ARF, however, differ from those for patients with chronic ren...
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ژورنال
عنوان ژورنال: Nephrology Dialysis Transplantation
سال: 2001
ISSN: 1460-2385,0931-0509
DOI: 10.1093/ndt/16.suppl_6.23